Cancer
Breast, Colon and Prostate
Breast, Colon and Prostate
IGF-1
Insulin-like growth factor 1 is a hormone that promotes cell development and growth - also development and growth of cancer cells.
Obesity, dairy and meat consumption stimulate production of IGF-1.
The discovery of the "breast cancer gene"—BRCA-1—in 1994 led many women to believe that breast cancer was hereditary—just bad luck. In some cases, it led to premature amputation (!). The most famous example is that of actress Angelina Jolie. Breasts removed.
Is that true? Just bad luck?
No. Only 3% of all breast cancers can be attributed to genetic predisposition. And even then, environmental factors determine whether the inherited gene will develop into cancer. But for various economic reasons, it's more advantageous to scare people, offer surgery, and prescribe medication.
The biggest risk factors for breast cancer are:
Early menstruation
Late menopause
High blood levels of female hormones
High blood cholesterol
A diet high in animal products and processed carbohydrates:
Promotes early menstruation
Increases the age of menopause
Increases blood levels of female hormones
Increases cholesterol levels
You can do the math right?
Estrogen exposure is almost three times higher in women who eat a lot of animal products, and it's the estrogen level that has a significant impact on the development of breast cancer. Because a dairy cow typically produces milk 300 days a year (completely against its nature), the estrogen dose is ridiculously high, and therefore also for the consumers of her breast milk: dairy consumers!
Colditz GA, Willett W, Hunter DJ, et al. “Family history, age, and risk of breast cancer. Prospective data from the Nurses’ Health Study.” JAMA 270 (1993): 338-343
Campbell, T.C., Campbell, T. M. The China Study. BenBella Books. Texas, USA. 2016. p. 150
Bernstein, L, and Ross RK. “Endogenous hormones and breast cancer risk.” Epidemiol. Revs. 15 (1993): 48-65
Pike MC, Spicer DV, Dahmoush L, et al. “Estrogens, progesterons, normal breast cell proliferation, and breast cancer risk.” Epidemiol. Revs. 15 (1993): 17-35
Colon cancer (colon/rectum = colorectal cancer) is much more common in "developed" countries than in less developed ones. This, again, has nothing to do with genetic predisposition, but rather with lifestyle, where diet plays the most significant role.
In the Western world, where a standard diet rich in animal products prevails, half the population will have developed a tumor in the colon by the age of 70, with 10% developing into a malignant tumor.
The single greatest risk factor for developing colon cancer is meat consumption. This undeniable evidence has been available for a long time. Furthermore, research has shown that a lack of fiber in the daily diet promotes colorectal cancer, while eating enough fiber reduces the risk of this cancer by 33%!
Kinzler KW1, Vogelstein B. Lessons from hereditary colorectal cancer. Cell. 1996 Oct 18;87(2):159-70.
Armstrong B, Doll R. Environmental factors and cancer incidence and mortality in different countries, with special reference to dietary practices. Int J Cancer. 1975 Apr 15;15(4):617-31.
Burkitt DP. Epidemiology of cancer of the colon and rectum. Cancer. 1971 Jul;28(1):3-13.
Prostate cancer is a very slow-developing cancer, and a man with prostate cancer is likely to die sooner from causes other than this cancer, but not always. Prostate cancer is most common in countries that follow a Western diet and lifestyle. Here too, dairy is the devil. Studies show that consuming more dairy doubles your risk of prostate cancer and quadruples the risk of a fatal form of prostate cancer, compared to men who consume little or no dairy.
June M. Chan and Edward L. Giovannucci Dairy Products, Calcium, and Vitamin D and Risk of Prostate Cancer Epidemiologic Reviews Copyright © 2001 by the Johns Hopkins University Bloomberg School of Public Health
Meng Yang,1 Stacey A. Kenfield,2 Erin L. Van Blarigan, et al. Dietary patterns after prostate cancer diagnosis in relation to disease-specific and total mortality. Cancer Prev Res (Phila). 2015 Jun; 8(6): 545–551.